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Official PDF TranslationActa Biochimica et Biophysica Sinica

Nicotinamide mononucleotide ameliorates ionizing radiation-induced spermatogenic dysfunction in mice by modulating the glycolytic pathway

Authors: Wenqin Yang; Weihua Nong; Ke Liu; Xiaocan Lei; Xiaping Chen; Pei Jiang; Jiayi Tang; Cong Hu; Zecheng Hu; Meixiang Li

DOI: 10.3724/abbs.2024167Status: Verified Translated Edition
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Key Findings in This Report

• NMN supplementation (500 mg/kg) protects against ionizing radiation-induced testicular injury and spermatogenic dysfunction in adult male mice. • NMN restores serum testosterone, LH, and FSH levels, as well as testicular NAD+, lactate, and pyruvate levels after 5 Gy γ-ray irradiation. • NMN downregulates proapoptotic genes (Bax, Caspase-3) and upregulates antiapoptotic Bcl-2, while enhancing Sertoli cell markers and glycolytic enzyme genes (HK2, PKM2, LDHA). • The protective mechanism likely involves promoting spermatogenic cell proliferation and activating glycolytic pathways, suggesting NMN as a potential radioprotector for male cancer patients.