miR-340-3p-modified bone marrow mesenchymal stem cell-derived exosomes inhibit ferroptosis through METTL3-mediated m6A modification of HMOX1 to promote recovery of injured rat uterus

• MB-exos suppress ferroptosis in endometrial stromal cells, promoting recovery of injured rat uterus. • MB-exos downregulate METTL3, reducing m6A modification levels and stabilizing HMOX1 mRNA to inhibit ferroptosis. • YTHDF2 is identified as the m6A reader that degrades HMOX1 mRNA, a key mechanism in ferroptosis regulation. • Targeting m6A modification and ferroptosis offers a novel therapeutic strategy for endometrial injury and intrauterine adhesions.