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Official PDF TranslationActa Biochimica et Biophysica Sinica

The mechanism by which piR-000699 targets SLC39A14 regulates ferroptosis in aging myocardial ischemia/reperfusion injury

Authors: Hongyang Chi; Yue’e Chai; Lingju Ma; Yichen Wang; Qianqian Wu; Lexin Wang; Junjie Zhai; Fufun Ma; Yancheng Tian; Ning Qi; Jianhong Peng; Youjuan Fu; Xiaoling Yang; Hui Huang; Shengchao Ma

DOI: 10.3724/abbs.2024024Status: Verified Translated Edition
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Key Findings in This Report

• piR-000699 is upregulated in aging cardiomyocytes under hypoxia/reoxygenation, promoting ferroptosis. • SLC39A14 is identified as a direct target of piR-000699, mediating ferroptosis in aging myocardial I/R injury. • Ferroptosis is a key cell death pathway in aging myocardial I/R injury, both in vivo and in vitro. • Targeting piR-000699/SLC39A14 axis may offer a novel therapeutic strategy for aged myocardial I/R injury.