🧬 SinoBioData Academic Portal
Official PDF TranslationStem Cell Research & Therapy

PTPN2 deficiency amplifies inflammatory signalling and impairs functional maturation of human stem cell-derived islets

Authors: Valerie Vandenbempt; Javier Negueruela; Francisco Ribeiro-Costa; Mariana Nunes; Leonardo Traini; Sema Elif Eski; Wadsen St-Pierre-Wijckmans; Stéphane Demine; Nicolas Baeyens; Hazem Ibrahim; Luciano G. Martelotto; Sumeet Pal Singh; Alessandra K. Cardozo; Mayank Bansal; Esteban N. Gurzov

DOI: 10.1186/s13287-025-04892-4Status: Verified Translated Edition
Sponsored AdvertisementAd Placement Area
reCAPTCHA Bot Shield Active

Preparing Secure Academic Download

Verifying human reader & generating high-resolution document...

Verifying Document Integrity15s remaining
← Back to Article
Protected by Google reCAPTCHA v3.PrivacyTerms
Sponsored ContentAdSense In-Feed Ad Slot

Key Findings in This Report

• PTPN2 expression is dynamically regulated during SC-islet differentiation and altered in T1D β cells, suggesting a role in β-cell stress responses. • PTPN2 deficiency amplifies interferon-induced inflammatory signalling and impairs SC-islet differentiation efficiency in vitro. • PTPN2-knockout SC-islets show reduced glycaemic control after implantation, linked to diminished endocrine identity and enhanced interferon signalling. • PTPN2 is a potential therapeutic target to preserve β-cell function and improve stem cell-derived islet maturation for diabetes cell replacement therapy.