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Official PDF TranslationActa Biochimica et Biophysica Sinica

METTL3-mediated m6A modification of pri-miRNA-31 promotes hypertrophic scar progression

Authors: Qirui Wang; Jialin Hou; Siyi Zeng; Xue Wang; Yimin Liang; Renpeng Zhou

DOI: 10.3724/abbs.2025033Status: Verified Translated Edition
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Key Findings in This Report

• METTL3 and m6A methylation are upregulated in hypertrophic scar tissues compared to normal skin, suggesting a role in HS pathogenesis. • Knockdown of METTL3 in HS fibroblasts reduces collagen and α-SMA expression, inhibits proliferation and migration, and induces G1 phase arrest. • METTL3 promotes maturation of pri-miR-31 to miR-31-5p via m6A modification, and miR-31-5p partially reverses the anti-fibrotic effects of METTL3 inhibition. • ZBTB20 is identified as a downstream target of miR-31-5p, and its knockdown inhibits fibroblast fibrosis, revealing a novel therapeutic target for HS.