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Official PDF TranslationChinese Journal of Tissue Engineering Research

Animal models of neurogenic heterotopic ossification: key disease progression and pathogenesis

Authors: Ren Qingsong; Xie Yulei; Liu Jingjing; Lin Jingyi; Long Danlei; Zhang Chunyu; Xie Liang; Zheng Kaiyuan; Wang Yinxu

DOI: 10.12307/2026.21299Status: Verified Translated Edition
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Key Findings in This Report

• Macrophage depletion reduces neurogenic heterotopic ossification volume by approximately 90%. • Fibro-adipogenic progenitor pathological reprogramming is driven by substance P, calcitonin gene-related peptide, oncostatin M/JAK/STAT, IL-1β, and lipopolysaccharide. • Hypoxia-angiogenesis signaling via HIF-1α/VEGF is a critical mechanism. • Animal models include spinal cord injury (cardiotoxin, denervation, tendon injury, lipopolysaccharide) and traumatic brain injury (free fall, controlled cortical impact, fluid percussion plus peripheral trauma).