SinoBioData Academic Portal
XZ
Verified CAS / Academic Author2 Decoded Studies

Prof. XU Zemei

Sun Yat-sen University

Research Publications & English Decoded Briefs

Showing 2 publications
Acta Biochimica et Biophysica Sinica2025DOI: 10.3724/abbs.2025099

The dual role of RNA-binding proteins: promotion of tumorigenesis, drug resistance, and emerging therapeutic targets

Cancer is a complex and multifaceted disease characterized by a multitude of molecular factors. RNA-binding proteins (RBPs) have emerged as pivotal regulators of tumor development, progression, and chemoresistance through their interactions with target transcripts. These interactions regulate a multitude of processes, including alternative splicing, cleavage and polyadenylation, RNA localization, translation, N6-methyladenosine (m6A) RNA modification, and DNA double-strand break repair. The RBP family comprises over 2000 proteins and plays a critical role in oncogene expression, invasion, metastasis, and inhibition of apoptosis. However, the mechanisms by which RBPs selectively recognize RNAs remain an active area of research. In this review, we examine recent advancements in understanding RNA-binding domains and the RNA processes regulated by RBPs in tumorigenesis, summarize and highlight the roles of RNA-binding domains in cancers and the molecular mechanisms of RBPs in chemotherapy resistance, discuss the potential of targeting RBPs for cancer therapy and review RBPs that are dysregulated in cancers. Additionally, we highlight recently developed tools for predicting RBP-RNA binding activities to provide valuable support for ongoing research efforts.

Acta Biochimica et Biophysica Sinica2025DOI: 10.3724/abbs.2025099

The dual role of RNA-binding proteins: promotion of tumorigenesis, drug resistance, and emerging therapeutic targets

RNA-binding proteins (RBPs) constitute a family of over 2000 proteins that regulate post-transcriptional gene expression through interactions with target transcripts. These interactions govern alternative splicing, cleavage and polyadenylation, RNA localization, translation, N6-methyladenosine (m6A) modification, and DNA double-strand break repair. RBPs are frequently dysregulated in cancers, where they modulate oncogene expression, invasion, metastasis, and apoptosis inhibition. This review examines recent advances in understanding RNA-binding domains (RBDs) and the RNA processes they regulate in tumorigenesis. We summarize the roles of six well-characterized RBDs in cancers and the molecular mechanisms by which RBPs contribute to chemotherapy resistance. Specific examples include PCBP2-mediated upregulation of UFD1 and NT5E in breast cancer, PABPN1 regulation of mRNA alternative polyadenylation in bladder cancer, and IGF2BP3-driven metabolic reprogramming conferring resistance to EGFR inhibitors in non-small cell lung cancer. We discuss the potential of targeting RBPs for cancer therapy and review RBPs that are dysregulated across malignancies. Additionally, we highlight recently developed tools for predicting RBP-RNA binding activities, including molecular dynamics simulation, nuclear magnetic resonance, and crystallography. These computational and structural approaches provide valuable support for ongoing research efforts to elucidate the mechanisms by which RBDs recognize RNA motifs, a process mediated by van der Waals interactions and hydrogen bonds. The clinical implications of RBP dysregulation underscore their prognostic significance and therapeutic potential as emerging targets in oncology.